Research

Hormonal

Long-acting GLP-1 receptor agonists (liraglutide and semaglutide) activate POMC neurons and inhibit NPY/AgRP neurons in vivo, with effects occurring 12-24 hours post-injection and dependent on the TRPC5 channel and metabolic state.

Long-acting GLP-1 drugs like liraglutide and semaglutide change brain activity related to hunger and energy use, but this happens slowly, peaking 12-24 hours after injection. This effect is stronger when you are fasting. The drug works by activating specific brain cells (POMC) that reduce hunger and inhibiting others (NPY/AgRP) that increase it, using a channel called TRPC5.

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We found that liraglutide and semaglutide directly activate and increase excitatory tone to POMC neurons in a time-dependent manner... Conversely, GLP-1Ra’s decreased excitatory input to and indirectly inhibited NPY/AgRP neurons through activation of K-ATP and TRPC5 channels in GABAergic neurons. Notably, the temporal activation of POMC and inhibition of NPY/AgRP neuronal activity after liraglutide or semaglutide was injected [either intraperitoneal (I.P.) or subcutaneous (S$C.)] was dependent upon the nutritional state of the animals (fed vs food-deprived).
Yanbin Dong et al. · Molecular Metabolism · 2021

Why this rating

High-quality in vivo mouse models with electrophysiology and fiber photometry, though not human clinical trials.

Source

Time and metabolic state-dependent effects of GLP-1R agonists on NPY/AgRP and POMC neuronal activity in vivo

Yanbin Dong et al. · Molecular Metabolism · 2021

DOI 10.1016/j.molmet.2021.101352

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DOI resolved against Crossref · corpus check 2026-06-10

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