Research

Hormonal

Genetically proxied activation of the glucagon-like peptide-1 receptor (GLP1RA) is causally associated with a reduced risk of schizophrenia.

This study suggests that medications activating the GLP-1 receptor (like semaglutide or liraglutide) may lower the risk of developing schizophrenia, primarily through their effect on body weight. For individuals at high risk of schizophrenia who are overweight, using these medications for weight management might offer a dual benefit of metabolic health and reduced psychiatric risk. However, this is based on genetic data, not direct clinical trials, so it should not replace standard psychiatric care.

GoodSupportsHIGH confidence
We found genetic evidence suggesting a causal relationship between GLP1RA and a reduced risk of schizophrenia [OR (95% CI) = 0.84 (0.71–0.98), I2 = 0.0%, common effects model].
Longgang Xiang et al. · International Journal of Molecular Sciences · 2025

Why this rating

Mendelian Randomization provides strong causal inference for genetic proxies, though it does not directly translate to clinical dosing.

Source

Impact of Glucagon-like Peptide-1 Receptor Agonists on Mental Illness: Evidence from a Mendelian Randomization Study

Longgang Xiang et al. · International Journal of Molecular Sciences · 2025

DOI 10.3390/ijms26062741

Meta-analysis · 2 studiesCited 5×
Read the paper
DOI resolved against Crossref · corpus check 2026-06-10

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