Research

Hormonal

Endogenous GIP acts as an incretin that potentiates glucose-stimulated insulin secretion, and its inhibition via GIP receptor antagonists reveals this physiological role.

GIP naturally helps your body release insulin after eating. Early treatments tried adding more GIP, but it didn't work well in diabetics. New research shows that blocking GIP's natural signal helps scientists understand its true role, which is still to boost insulin, even in diabetes.

StrongSupportsVERY_HIGH confidence
GIP is an incretin hormone that, along with glucagon-like peptide 1 (GLP-1), postprandially potentiates glucose-stimulated insulin secretion from pancreatic beta cells (16).
Frederikke Koefoed-Hansen et al. · Frontiers in Endocrinology · 2025

Why this rating

Well-established physiology supported by multiple citations and consistent findings across studies.

Source

The evolution of the therapeutic concept ‘GIP receptor antagonism’

Frederikke Koefoed-Hansen et al. · Frontiers in Endocrinology · 2025

DOI 10.3389/fendo.2025.1570603

narrative_reviewCited 4×
Read the paper
DOI resolved against Crossref · corpus check 2026-06-10

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