Research

Hormonal

Human obesity is characterized by tissue-specific dysregulation of cortisol metabolism, specifically involving increased 11beta-hydroxysteroid dehydrogenase type 1 (11beta-HSD1) activity in adipose tissue and skeletal muscle, leading to elevated intracellular cortisol levels despite normal circulating cortisol.

Standard blood tests for cortisol may appear normal in obesity, masking tissue-specific metabolic stress. The problem lies in how fat and muscle cells convert inactive cortisone into active cortisol via the enzyme 11beta-HSD1. Managing obesity requires addressing this local tissue dysregulation, not just systemic stress markers.

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We have shown that 11beta-HSD1 activity is increased in adipose tissue and skeletal muscle in human obesity, leading to increased intracellular cortisol concentrations.
Eva Rask et al. · The Journal of Clinical Endocrinology & Metabolism · 2001

Why this rating

The study uses rigorous biochemical assays (microsomal fractions) and direct tissue biopsies in humans, though it is a cross-sectional observation rather than a long-term intervention.

Source

Tissue-Specific Dysregulation of Cortisol Metabolism in Human Obesity

Eva Rask et al. · The Journal of Clinical Endocrinology & Metabolism · 2001

DOI 10.1210/jcem.86.3.7453

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DOI resolved against Crossref · corpus check 2026-06-10

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