Research

Hormonal

Administration of the intestine-restricted FXR agonist fexaramine (FEX) improves glucose tolerance and promotes adipose tissue browning in mice by reshaping gut microbiota to increase lithocholic acid (LCA) production, which activates TGR5/GLP-1 signaling.

This research suggests that specific bile acid signaling, influenced by gut bacteria, plays a key role in how your body handles sugar and fat storage. While this study used a specific drug (fexaramine) in mice, it highlights the importance of gut health in metabolic regulation. For now, there is no direct human supplement equivalent, but maintaining a diverse gut microbiome through diet is a logical step to support these natural pathways.

GoodSupportsHIGH confidence
This study uncovered a mechanism in which activation of intestinal FXR shaped the gut microbiota to activate TGR5/GLP-1 signaling to improve hepatic glucose and insulin sensitivity and increase adipose tissue browning; the gut microbiota plays a critical role in bile acid metabolism and signaling to regulate metabolic homeostasis in health and disease.
Preeti Pathak et al. · Hepatology · 2018

Why this rating

The study uses rigorous genetic knockout models (Fxr-/-, Tgr5-/-) and antibiotic reversal to establish causality, though it is conducted in mice.

Source

Intestine farnesoid X receptor agonist and the gut microbiota activate G‐protein bile acid receptor‐1 signaling to improve metabolism

Preeti Pathak et al. · Hepatology · 2018

DOI 10.1002/hep.29857

mechanism_only · n=20Cited 535×
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DOI resolved against Crossref · corpus check 2026-06-10

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