Research

Hormonal

Oral administration of the intestine-selective FXR inhibitor Gly-MCA prevents and reverses high-fat diet-induced and genetic obesity, insulin resistance, and hepatic steatosis in mice by reducing intestinal ceramide biosynthesis, which restores beige fat thermogenic function.

This research identifies a specific bile acid derivative (Gly-MCA) that, when taken orally, blocks a specific receptor (FXR) in the gut. This blockage reduces harmful fats (ceramides) in the intestine, which allows body fat to burn energy more effectively, leading to weight loss and better blood sugar control in obese mice. While promising, this is currently a preclinical finding in mice and not yet an available human treatment.

ModerateSupportsMEDIUM confidence
Gly-MCA is a selective high-affinity FXR inhibitor that can be administered orally and prevents, or reverses, high-fat diet-induced and genetic obesity, insulin resistance and hepatic steatosis in mice. Mechanistically, the metabolic improvements with Gly-MCA depend on reduced biosynthesis of intestinal-derived ceramides, which directly compromise beige fat thermogenic function.
Changtao Jiang et al. · Nature Communications · 2015

Why this rating

High-quality mechanistic data in mouse models, but lacks human clinical trial data for efficacy and safety.

Source

Intestine-selective farnesoid X receptor inhibition improves obesity-related metabolic dysfunction

Changtao Jiang et al. · Nature Communications · 2015

DOI 10.1038/ncomms10166

mechanism_only · n=66Cited 535×
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DOI resolved against Crossref · corpus check 2026-06-10

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