Research

Hormonal

Twelve-week treatment with the GLP-1 analogue liraglutide (1.8 mg/day) reduces hepatic de novo lipogenesis (DNL) and improves adipose tissue insulin sensitivity, thereby decreasing lipotoxicity in patients with biopsy-proven non-alcoholic steatohepatitis (NASH).

For patients with NASH, a 12-week course of liraglutide (titrated to 1.8 mg daily) has been shown to directly reduce the liver's production of new fat (DNL) and improve how fat tissue responds to insulin. This dual action reduces the toxic fat spill-over (lipotoxicity) that drives liver damage. While weight loss occurs, the study suggests direct metabolic benefits on the liver and fat tissue independent of weight change alone.

GoodSupportsHIGH confidence
Liraglutide reduced hepatic DNL in-vivo (-1.26 vs. +1.30 %; p<0.05)... Liraglutide increased adipose tissue insulin sensitivity enhancing the ability of insulin to suppress lipolysis both globally... and specifically within subcutaneous adipose tissue (p<0.05).
Matthew J. Armstrong et al. · Journal of Hepatology · 2015

Why this rating

Randomized, double-blind, placebo-controlled trial with mechanistic depth (clamps, isotopes, in vitro), though limited by small sample size (n=14).

Source

Glucagon-like peptide 1 decreases lipotoxicity in non-alcoholic steatohepatitis

Matthew J. Armstrong et al. · Journal of Hepatology · 2015

DOI 10.1016/j.jhep.2015.08.038

rct · n=14Cited 437×
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DOI resolved against Crossref · corpus check 2026-06-10

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