Research

Hormonal

In Type 2 Diabetes, elevated circulating levels of microRNA-144 (miR-144) directly inhibit the expression of Insulin Receptor Substrate 1 (IRS1), thereby impairing insulin signaling and contributing to insulin resistance.

This research identifies a specific molecular mechanism in Type 2 Diabetes where high levels of miR-144 block insulin signaling by reducing IRS1. For patients, this highlights that insulin resistance is driven by specific genetic regulatory failures, not just lifestyle factors. While this paper does not offer a direct treatment, it identifies miR-144 as a potential biomarker for disease severity and a target for future therapies aimed at restoring insulin sensitivity.

GoodSupportsHIGH confidence
Increased circulating level of miR-144 has been found to correlate with down-regulation of its predicted target, insulin receptor substrate 1 (IRS1) at both mRNA and protein levels. We could also experimentally demonstrate that IRS1 is indeed the target of miR-144.
Dwi Setyowati Karolina et al. · PLoS ONE · 2011

Why this rating

Strong evidence combining human observational data (correlation in patients), animal models (rat tissues), and in vitro mechanistic validation (luciferase assays, cell transfection).

Source

MicroRNA 144 Impairs Insulin Signaling by Inhibiting the Expression of Insulin Receptor Substrate 1 in Type 2 Diabetes Mellitus

Dwi Setyowati Karolina et al. · PLoS ONE · 2011

DOI 10.1371/journal.pone.0022839

mechanism_only · n=50Cited 429×
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DOI resolved against Crossref · corpus check 2026-06-10

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