Research

Hormonal

High-fructose diets promote visceral adipose tissue accumulation and insulin resistance, which indirectly drives intrahepatic lipid accumulation by increasing the flux of free fatty acids from adipose tissue to the liver.

Fructose may contribute to belly fat (visceral adiposity) more than glucose in some studies. This belly fat releases fatty acids into the liver, worsening fatty liver. This is an indirect effect. Managing overall weight and insulin sensitivity is key to reducing this flux.

ModerateQualifiesMEDIUM confidence
In one 10-week diet-intervention trial, the consumption of fructose, but not glucose, promoted the accumulation of visceral adipose tissue... Thus, it remains debated whether fructose consumption directly influences adipocyte lipolysis and/or visceral adiposity, but it may contribute to an increase in adipose-to-liver FA trafficking through several indirect mechanisms.
Kasper W. ter Horst et al. · Nutrients · 2017

Why this rating

Evidence is mixed; some trials show visceral fat gain, others do not, and direct lipolysis effects are unclear.

Source

Fructose Consumption, Lipogenesis, and Non-Alcoholic Fatty Liver Disease

Kasper W. ter Horst et al. · Nutrients · 2017

DOI 10.3390/nu9090981

narrative_reviewCited 357×
Read the paper
DOI resolved against Crossref · corpus check 2026-06-10

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