Research

Hormonal

Mitochondrial dysfunction is a necessary driver of the Senescence-Associated Secretory Phenotype (SASP), as evidenced by the elimination of SASP in cells devoid of mitochondria, while irreversible cell cycle arrest persists.

Aging and age-related inflammation are driven by dysfunctional mitochondria producing the SASP. Interventions that target mitochondrial health (e.g., senolytics, senostatics like rapamycin or metformin) may reduce this inflammatory burden. Focus on strategies that support mitochondrial quality control, such as exercise and metabolic health, rather than just energy output.

GoodSupportsHIGH confidence
Using this tool we showed that senescent cells which contained no mitochondria had no SASP (both at protein and mRNA level) but still underwent the irreversible cell cycle arrest [71].
James Chapman et al. · FEBS Letters · 2019

Why this rating

Based on a review of multiple studies including proof-of-principle experiments using PINK1/Parkin-induced mitophagy.

Source

Mitochondrial dysfunction and cell senescence: deciphering a complex relationship

James Chapman et al. · FEBS Letters · 2019

DOI 10.1002/1873-3468.13498

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DOI resolved against Crossref · corpus check 2026-06-10

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