Research

Hormonal

Reduced signaling of the Growth Hormone/Insulin-Like Growth Factor (GH/IGF-I) system extends longevity and delays aging in mammals, primarily through enhanced insulin sensitivity, reduced oxidative stress, and increased stress resistance.

Current research in long-lived mouse models suggests that lower levels of Growth Hormone and IGF-I signaling are associated with extended longevity and delayed aging. While GH therapy is marketed for anti-aging, genetic evidence points to reduced signaling as the beneficial pathway. This does not currently translate to a recommended intervention for healthy humans, but highlights the importance of metabolic health and insulin sensitivity.

GoodSupportsHIGH confidence
In the mouse, several spontaneous or experimentally induced mutations that interfere with GH biosynthesis, GH actions, or sensitivity to IGF-I lead to extended longevity... Extended longevity of hypopituitary and GH-resistant mice appears to be due to multiple mechanisms including reduced insulin levels, enhanced insulin sensitivity, alterations in carbohydrate and lipid metabolism, reduced generation of reactive oxygen species, enhanced resistance to stress, reduced oxidative damage, and delayed onset of age-related disease.
Andrzej Bartke · Endocrinology · 2005

Why this rating

Strong evidence from multiple genetically modified mouse models (Ames, Snell, GHRKO) and supporting human epidemiological data (centenarians), though direct human intervention trials are not the primary focus.

Source

Minireview: Role of the Growth Hormone/Insulin-Like Growth Factor System in Mammalian Aging

Andrzej Bartke · Endocrinology · 2005

DOI 10.1210/en.2005-0411

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DOI resolved against Crossref · corpus check 2026-06-10

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