Research

Hormonal

Selective leptin resistance in obesity preserves the sympathoexcitatory effects of leptin (increasing blood pressure) while blunting its appetite-suppressing effects.

In obesity, the body may become resistant to leptin's ability to tell you to stop eating, but it remains sensitive to leptin's ability to raise blood pressure. This explains why obese individuals often have high blood pressure even if they don't feel hungry.

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Mark et al. suggested that some forms of obesity may be characterized by a 'selective leptin resistance,' limited to its favorable metabolic effects (satiety and weight loss), while its sympathoexcitatory effects on the cardiovascular system are maintained.
Daniela Guarino et al. · Frontiers in Physiology · 2017

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Cited experimental and human studies.

Source

The Role of the Autonomic Nervous System in the Pathophysiology of Obesity

Daniela Guarino et al. · Frontiers in Physiology · 2017

DOI 10.3389/fphys.2017.00665

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DOI resolved against Crossref · corpus check 2026-06-10

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