Research

Hormonal

Deficiency or inhibition of the nuclear receptor RORγ1 protects against diet-induced insulin resistance and improves glucose tolerance, potentially through mechanisms distinct from RORα.

This research suggests that modulating RORγ1 activity could be a strategy to improve insulin sensitivity. The paper notes that RORγ1 expression positively correlates with adipocyte size and insulin resistance in humans, implying that inhibiting this receptor might help manage type 2 diabetes risk.

ModerateSupportsMEDIUM confidence
Deficiency in RORγ also protects against diet-induced insulin resistance by a mechanism that appears different from that in RORα deficiency... In obese RORγ −/− mice... Fasting blood insulin levels were shown to be significantly lower... and mice displayed improved insulin sensitivity.
Anton M. Jetten et al. · Frontiers in Endocrinology · 2013

Why this rating

Evidence is derived from mouse models (RORγ-deficient) and human observational studies correlating RORγ1 expression with adipocyte size and insulin resistance.

Source

Retinoic acid-related orphan receptors α and γ: key regulators of lipid/glucose metabolism, inflammation, and insulin sensitivity

Anton M. Jetten et al. · Frontiers in Endocrinology · 2013

DOI 10.3389/fendo.2013.00001

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DOI resolved against Crossref · corpus check 2026-06-10

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