Research

Hormonal

Postprandial administration of para-tyramine-O-sulphate (pTOS) acts as a conserved anorexigenic gut-brain signal that suppresses food intake and body weight in mice by activating ventromedial hypothalamus (VMH) neurons, without affecting energy expenditure or gastric emptying.

This research identifies pTOS, a metabolite produced by gut bacteria from dietary tyrosine, as a natural signal that tells your brain to stop eating. In mice, supplementing with pTOS reduced food intake and body weight without changing how much energy they burned or how fast their stomach emptied. The effect works by activating specific neurons in the hypothalamus. While human studies show pTOS levels rise after meals, the clinical application for weight loss is still in early stages, particularly for those with diabetes.

GoodSupportsHIGH confidence
Together, these data uncover a conserved postprandial anorexigenic metabolite that links nutrient intake to energy balance.
Shuke Xiao et al. · Nature Metabolism · 2026

Why this rating

Strong mechanistic evidence in mice (genetic silencing, electrophysiology) and observational data in humans/pythons, but human clinical efficacy data is limited to observational meal tests.

Source

Python metabolomics uncovers a conserved postprandial metabolite and gut–brain feeding pathway

Shuke Xiao et al. · Nature Metabolism · 2026

DOI 10.1038/s42255-026-01485-0

mechanism_onlyCited 1×
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DOI resolved against Crossref · corpus check 2026-06-10

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