Research

Hormonal

Vagal afferent signaling mediates nutrient-specific learning and satiation, and its blunting in obesity contributes to excessive weight gain, while targeted neuromodulation (stimulation or blockade) can reduce food intake and body weight.

Your gut communicates directly with your brain via the vagus nerve to regulate hunger and satiety. In obesity, this signal can become blunted. Therapies that stimulate this nerve (like non-invasive vagus nerve stimulation) or mimic gut hormones (like GLP-1 agonists) can help restore this signaling, reducing food intake and aiding weight loss. Focus on strategies that support gut-brain communication.

GoodSupportsHIGH confidence
Given that the sensitivity of gut vagal afferents is decreased in obese rodents (108), it will be important to further distinguish what comes first: obesity, or blunting of vagal afferent sensitivity (108).
Hans‐Rudolf Berthoud et al. · Journal of Clinical Investigation · 2021

Why this rating

The paper is a comprehensive review citing numerous animal studies and clinical trials, though it notes controversies and the need for more specific tools.

Source

Gut-brain communication and obesity: understanding functions of the vagus nerve

Hans‐Rudolf Berthoud et al. · Journal of Clinical Investigation · 2021

DOI 10.1172/jci143770

narrative_reviewCited 99×
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DOI resolved against Crossref · corpus check 2026-06-10

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