Research

Hormonal

Discontinuation of incretin-based pharmacotherapy (GLP-1 or dual GLP-1/GIP agonists) leads to partial or complete weight regain due to the reactivation of homeostatic neurohormonal systems (increased ghrelin, decreased leptin and peptide YY), confirming obesity as a chronic condition requiring long-term management.

If you stop taking your incretin medication (like semaglutide or tirzepatide), your body's natural hunger signals will likely return, causing you to regain the weight you lost. This is a biological response, not a lack of willpower. To keep the weight off, you must treat obesity as a chronic condition and continue your medication and lifestyle habits long-term, just as you would for high blood pressure.

StrongRefutesVERY_HIGH confidence
Evidence shows that cessation of GLP-1 receptor agonist therapy leads to partial or complete weight regain, underscoring the need for continued treatment and comprehensive management... Mechanistically, discontinuation leads to reactivation of homeostatic neurohormonal systems that favor weight regain—such as increased ghrelin and decreased leptin and peptide YY levels—highlighting the physiological drive to restore energy balance.
Natalia Senatorska et al. · International Journal of Innovative Technologies in Social Science · 2025

Why this rating

Supported by pivotal phase 3 randomized controlled trials (STEP 4, SURMOUNT-4) with robust long-term follow-up data.

Source

INCRETIN-BASED THERAPIES FOR OBESITY MANAGEMENT: THE IMPACT OF PATIENT EDUCATION AND LIFESTYLE MODIFICATION ON LONG-TERM TREATMENT OUTCOMES

Natalia Senatorska et al. · International Journal of Innovative Technologies in Social Science · 2025

DOI 10.31435/ijitss.4(48).2025.4225

narrative_review
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DOI resolved against Crossref · corpus check 2026-06-10

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