Research

Hormonal

Deletion of the p38α kinase in adipose tissue protects against high-fat diet-induced obesity by increasing energy expenditure and brown adipose tissue (BAT) thermogenesis.

Current research suggests that low levels of the p38α protein in fat tissue are associated with higher body mass index in humans. In mice, removing p38α from fat cells prevents obesity by boosting heat production. This implies that therapies aiming to reduce p38α activity in fat might help treat obesity, but this is currently only a mechanistic finding in animals, not a human treatment protocol.

GoodSupportsHIGH confidence
we unexpectedly found that lack of p38α protected against high-fat diet (HFD)-induced obesity. We also showed that p38αFab-KO mice presented higher energy expenditure due to increased BAT thermogenesis.
Nuria Matesanz et al. · PLoS Biology · 2018

Why this rating

Strong evidence from multiple genetically modified mouse models (p38αFab-KO, p38αUCP1-KO) and human tissue correlation, though human intervention data is absent.

Source

p38α blocks brown adipose tissue thermogenesis through p38δ inhibition

Nuria Matesanz et al. · PLoS Biology · 2018

DOI 10.1371/journal.pbio.2004455

mechanism_onlyCited 47×
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DOI resolved against Crossref · corpus check 2026-06-10

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