Research

Hormonal

Cry2 is critical for the circadian regulation of myogenic differentiation and fusion by maintaining Cyclin D1 expression and Tmem176b rhythmicity.

Cry2 helps coordinate the timing of muscle fiber fusion after injury. It ensures that cells don't exit the cell cycle too early and maintains the expression of genes needed for fibers to merge. Disruption of this clock component can lead to incomplete regeneration.

ModerateSupportsMEDIUM confidence
These defects in differentiation and regeneration processes in Cry2 KO animals and cell lines were attributed to a detrimental and premature exit from the cell cycle due to inhibited Cyclin D1 expression (28).
Ryan E. Kahn et al. · American Journal of Physiology-Cell Physiology · 2023

Why this rating

Based on Cry2 KO mice and cell lines; mechanism is detailed but requires validation in human physiological models.

Source

Molecular clocks, satellite cells, and skeletal muscle regeneration

Ryan E. Kahn et al. · American Journal of Physiology-Cell Physiology · 2023

DOI 10.1152/ajpcell.00073.2023

narrative_reviewCited 19×
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DOI resolved against Crossref · corpus check 2026-06-10

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