Research

Energy balance

In diabetic organs, mitochondrial superoxide production and oxidative phosphorylation are reduced, not increased, and this reduction is associated with organ dysfunction.

Understanding that diabetic damage may stem from mitochondrial 'shutdown' rather than 'overdrive' helps explain why simple antioxidant supplements haven't worked. The focus shifts to restoring mitochondrial activity through lifestyle changes.

ModerateRefutesMEDIUM confidence
Recent data suggest that in response to excess glucose exposure or nutrient stress, there is a reduction of mitochondrial superoxide, oxidative phosphorylation, and mitochondrial ATP generation in several target tissues of diabetes complications.
Kumar Sharma · Diabetes · 2015

Why this rating

Based on animal models (STZ, Akita mice) and in vitro studies; in vivo human data is limited.

Source

Mitochondrial Hormesis and Diabetic Complications

Kumar Sharma · Diabetes · 2015

DOI 10.2337/db14-0874

narrative_reviewCited 188×
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DOI resolved against Crossref · corpus check 2026-06-10

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