Research

Mixed

Chronic oxidative stress and the resulting accumulation of oxidized, carbonylated proteins drive the loss of proteostasis and trigger cellular senescence, a primary mechanism of aging.

Focus on reducing chronic oxidative stress through a nutrient-dense diet rich in antioxidants (micronutrients, trace elements) and maintaining metabolic health to support proteostasis. This may help delay the onset of cellular senescence and age-related diseases.

GoodSupportsHIGH confidence
Aging is characterized by the accumulation of cellular damage, in turn, leading to increased susceptibility to diseases... One major contributor is the development of cellular senescence... If oxidative stress persists, cellular senescence is a likely outcome... The risk for their accumulation lies in the formation of insoluble protein aggregates... The loss of proteostasis is therefore a likely and strong contributor to senescence and aging.
Annika Höhn et al. · Redox Biology · 2016

Why this rating

High-quality review of multiple lines of evidence including human clinical data, animal models, and cellular studies, though it is a review rather than a primary trial.

Source

Happily (n)ever after: Aging in the context of oxidative stress, proteostasis loss and cellular senescence

Annika Höhn et al. · Redox Biology · 2016

DOI 10.1016/j.redox.2016.12.001

narrative_reviewCited 357×
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DOI resolved against Crossref · corpus check 2026-06-10

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