Research

Hormonal

MCP-1 (CCL2) secretion from hypertrophied adipocytes recruits monocytes to adipose tissue, leading to M1 macrophage infiltration and insulin resistance.

High levels of MCP-1 in fat tissue act as a beacon, pulling inflammatory cells into fat. Reducing MCP-1 levels might help prevent this inflammatory infiltration.

GoodSupportsHIGH confidence
MCP-1 secreted from enlarged adipocytes attracted circulating monocytes to adipose tissue, causing inflammatory characteristics of adipose tissue.
Sanshiro Tateya et al. · Frontiers in Endocrinology · 2013

Why this rating

Supported by multiple studies cited in the review.

Source

Recent Advances in Obesity-Induced Inflammation and Insulin Resistance

Sanshiro Tateya et al. · Frontiers in Endocrinology · 2013

DOI 10.3389/fendo.2013.00093

narrative_reviewCited 210×
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DOI resolved against Crossref · corpus check 2026-06-10

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