Research

Hormonal

Resistance exercise stimulates muscle hypertrophy primarily through the activation of the mTOR pathway via PKB, while endurance exercise stimulates mitochondrial biogenesis primarily through AMPK activation of PGC-1alpha.

Understanding that strength and endurance use different molecular switches helps explain why you cannot maximize both simultaneously. To build muscle, focus on high-intensity resistance to activate PKB/mTOR. To improve endurance, focus on duration to activate AMPK/PGC-1alpha.

StrongSupportsVERY_HIGH confidence
Protein kinase B/Akt can both activate protein synthesis and decrease protein breakdown, thus leading to hypertrophy, and AMP-activated protein kinase can increase mitochondrial protein, glucose transport, and a number of other factors that result in an endurance phenotype.
Keith Baar · Medicine & Science in Sports & Exercise · 2006

Why this rating

The paper cites multiple studies confirming these pathways in various models (chickens, rabbits, rats, humans).

Source

Training for Endurance and Strength

Keith Baar · Medicine & Science in Sports & Exercise · 2006

DOI 10.1249/01.mss.0000233799.62153.19

narrative_reviewCited 179×
Read the paper
DOI resolved against Crossref · corpus check 2026-06-10

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