Research

Hormonal

Alternative splicing of the p53 gene produces the p44 variant, which accelerates aging phenotypes, cognitive decline, and senescence by hyperactivating the IGF-1 pathway.

Current research focuses on molecular mechanisms; no direct human intervention is recommended based solely on this paper.

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overexpression of p44 in transgenic mice (p44+/+) leads to an accelerated age-associated phenotype, suppresses cell proliferation, and increases senescence... p44+/+ transgenic mice also display premature synaptic deficit, cognitive decline as well as Alzheimer’s disease-like features
Mathieu Deschênes et al. · Aging Cell · 2017

Why this rating

Supported by transgenic mouse models (Maier et al., 2004; Pehar et al., 2010, 2014).

Source

The emerging role of alternative splicing in senescence and aging

Mathieu Deschênes et al. · Aging Cell · 2017

DOI 10.1111/acel.12646

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DOI resolved against Crossref · corpus check 2026-06-10

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