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Chronic exposure to high glucose concentrations inhibits AMPK activity, potentially as a protective mechanism to limit glucose uptake and prevent oxidative stress during hyperglycemia.

High glucose levels inhibit AMPK activity. This inhibition may serve as a protective mechanism to limit further glucose uptake and prevent oxidative stress. Maintaining stable blood glucose levels helps preserve healthy AMPK function.

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AMPK can be negatively regulated by chronic exposure to high glucose. Acute hyperglycemia reduces AMPK activation in muscle, liver (Kraegen et al., 2006) and kidney (Lee, MJ et al., 2007).
Benoı̂t Viollet et al. · Critical Reviews in Biochemistry and Molecular Biology · 2010

Why this rating

Multiple studies in muscle, liver, and kidney show consistent inhibition.

Source

AMPK inhibition in health and disease

Benoı̂t Viollet et al. · Critical Reviews in Biochemistry and Molecular Biology · 2010

DOI 10.3109/10409238.2010.488215

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DOI resolved against Crossref · corpus check 2026-06-10

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