Research
Cellular
Semaglutide improves the function and inflammatory status of endothelial progenitor cells (EPCs) by inhibiting LPS-induced macrophage expression of miR-155 in exosomes.
Semaglutide may be beneficial in improving endothelial function in obesity-related inflammation.
StrongSupportsmedium confidence
Semaglutide improved the cell viability of EPCs and also inhibited the expression of inflammatory factors in EPCs as well as miR-155 in exosomes.
Why this rating
The study design involves direct experimental interventions and observations.
Source
Semaglutide alleviates inflammation-Induced endothelial progenitor cells injury by inhibiting MiR-155 expression in macrophage exosomes
Xiaoyu Pan et al. · International Immunopharmacology · 2023
DOI 10.1016/j.intimp.2023.110196
otherCited 27×
Read the paper DOI resolved against Crossref · corpus check 2026-06-10
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